Optimization of a Dicarboxylic Series for in Vivo Inhibition of Citrate Transport by the Solute Carrier 13 (SLC13) Family.

نویسندگان

  • Kim Huard
  • James R Gosset
  • Justin I Montgomery
  • Adam Gilbert
  • Matthew M Hayward
  • Thomas V Magee
  • Shawn Cabral
  • Daniel P Uccello
  • Kevin Bahnck
  • Janice Brown
  • Julie Purkal
  • Matthew Gorgoglione
  • Adhiraj Lanba
  • Kentaro Futatsugi
  • Michael Herr
  • Nathan E Genung
  • Gary Aspnes
  • Jana Polivkova
  • Carmen N Garcia-Irizarry
  • Qifang Li
  • Daniel Canterbury
  • Mark Niosi
  • Nicholas B Vera
  • Zhenhong Li
  • Bhagyashree Khunte
  • Jaclyn Siderewicz
  • Timothy Rolph
  • Derek M Erion
چکیده

Inhibition of the sodium-coupled citrate transporter (NaCT or SLC13A5) has been proposed as a new therapeutic approach for prevention and treatment of metabolic diseases. In a previous report, we discovered dicarboxylate 1a (PF-06649298) which inhibits the transport of citrate in in vitro and in vivo settings via a specific interaction with NaCT. Herein, we report the optimization of this series leading to 4a (PF-06761281), a more potent inhibitor with suitable in vivo pharmacokinetic profile for assessment of in vivo pharmacodynamics. Compound 4a was used to demonstrate dose-dependent inhibition of radioactive [(14)C]citrate uptake in liver and kidney in vivo, resulting in modest reductions in plasma glucose concentrations.

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عنوان ژورنال:
  • Journal of medicinal chemistry

دوره 59 3  شماره 

صفحات  -

تاریخ انتشار 2016